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New Product: Easy Heat

Posted on Tuesday, December 20, 2016

Gutta Percha Obturation Apical Condensation

System B compatible cordless

Easy to operate, rapid heating button pressing = heating, release the button for cooling.

The Easy Heat unit is wireless with high technology charging technology, stylish, fashionable. Tip temperature can be set to low temperature 160-200ºC and up to high temperature of 200-240ºC Easy Heat tips are sterilized and can be repeatedly used.

Pack Contains

·       Obturation Pen

·       Charging Base

·       Adaptor

·       Tips (4pcs)

·       User Guide

To order or for more information please call your supply house, or Tel. 1-800-454-9244  / 

3M Hosts Oral Health Council

Posted on Tuesday, December 20, 2016

In too many places around the world, people are suffering immensely from poor oral health. Despite its critical role in overall health, oral care is often overlooked, resulting in a population that suffers. Children who aren’t given proper access to care often have caries and struggle in school because they are in so much pain. These children grow up to become adults and elderly patients who aren’t nearly as educated or healthy as they could be. As a step to ignite worldwide change for oral health, 3M welcomed professionals and policy makers from around the world to the first-ever Oral Health Council on November 28-30 at the 3M Innovation Center.


The meeting served as a forum for industry leaders from Chile, China, Bolivia, Colombia, the United Kingdom, Peru, Russia, and the United States to share the challenges their countries face in delivering care to patients. Attendees learned from industry experts about preventive science innovations; how to better care for elderly, special-needs, and pediatric patients; and how to implement effective policy for oral health. Attendees left the meeting with solid action plans to improve oral health in their communities.


“Governments are instrumental in influencing the standard for health care, so people with a voice must advocate for change–even if it starts small at first,” says Simon Hearne, International Vice President for 3M Oral Care. “As a global industry leader, 3M is taking the responsibility to help people who can inspire change create a network that will aid them in promoting preventive oral care within their communities.”

3M says it plans to continue this global initiative and looks forward to the impact this council will have on the future of worldwide oral health.

Sonendo Closes $35 Million Equity Financing Round

Posted on Tuesday, December 20, 2016

Sonendo, Inc., the developer of a breakthrough technology for dentistry, announced today it has completed an equity financing totaling $35 million through new and existing investors.  The financing round led by DiNovA Capital will allow Sonendo to advance the commercialization of its GentleWave® System as an alternative to standard root canal treatment and develop additional products in adjacent verticals to leverage its Multisonic Ultracleaning™ technology platform. 

New investors DiNovA Capital and CVF, LLC, an affiliate of Henry Crown & Company, joined with existing investors Fjord Ventures, OrbiMed Advisors, LLC, Meritech Capital Partners, NeoMed Management and SEB Private Equity to form the investor syndicate.  In connection with the financing, Michael Zhao of DiNovA Capital has joined the Sonendo Board of Directors.  Mr. Zhao noted, “Sonendo’s GentleWave System is uniquely positioned to transform endodontic therapy by delivering exponential improvements in clinical efficacy.  DiNovA Capital is pleased to join Sonendo’s existing investors and we look forward to supporting the management and employees of Sonendo in bringing this exciting, innovative product to patients.”

Sonendo President and CEO Bjarne Bergheim commented, "With DiNovA Capital and CVF, LLC, we are pleased to expand our top-tier syndicate of committed, long-term investors who recognize the significant potential of this breakthrough technology.  Proceeds from this financing will provide the critical resources to support the growth of our commercialization efforts of the GentleWave System as well as continuing our development activities in furthering our Multisonic Ultracleaning technology platform.  Our investors share our passion for helping dentists deliver significant, patient-centric, improvements in clinical efficacy and treatment efficiency as we continue to live up to our mission of Saving Teeth Through Sound Science®.”

ADA: EPA Regulation on Dental Amalgam Waste Fair and Reasonable

Posted on Friday, December 16, 2016

CHICAGO — The American Dental Association (ADA) believes the Environmental Protection Agency’s (EPA) new federal regulation represents a fair and reasonable approach to the management of dental amalgam waste. The rule, released Dec. 15, requires dental practices nationwide to install devices, called separators, to capture dental amalgam waste preventing its release to sewer systems. The rule includes reasonable exemptions, a phase-in period and considerations for dental practices that have already installed the devices. 

The ADA shares the EPA’s goal of ensuring that dental amalgam waste is captured so that it may be recycled. The ADA originally commented on the draft regulation in February 2015. ADA representatives also met with the EPA and shared views and data as the process evolved. We commend the EPA for its consideration of the ADA’s comments.  

We believe this new rule—which is a federal standard—is preferable to a patchwork of rules and regulations across various states and localities.

Dental amalgam is a safe, affordable and durable filling material comprised of a mixture of metals, including silver, copper, tin and mercury. Although less than one percent of mercury released to the environment from man-made sources comes from dentistry, the ADA has long recognized the importance of dentists being good stewards of the environment by capturing and recycling dental amalgam. 

In 2002, long prior to federal regulation, the ADA published a set of voluntary guidelines for dentists on the capture and recycling dental amalgam. In 2007, the ADA added to its guidelines the use of amalgam separators that comply with the International Organization for Standardization’s (ISO) Standard 11143. The ADA is pleased to see that a number of provisions from the ADA guidelines are part of the new regulation.  

The ADA is in the process of reviewing the rule and background statement in detail, which is more than 90 pages in length, in order to develop practical resources which will be posted on to aid member dentists with questions they may have regarding compliance. In addition, ADA’s Business Resources has partnered with HealthFirst, a vendor that offers ADA member dentists special pricing on an amalgam separator device that will meet the federal regulatory requirements along with recycling services.


Tufts Dental School Honors Trailblazers

Posted on Friday, December 16, 2016

BOSTON (Dec. 16, 2016)--Two legends in the Tufts and dental medicine communities will be honored with Dean's Medals from Tufts University School of Dental Medicine at a ceremony today. The medals celebrate the contributions Hilde H. Tillman, DMD, and Esther M. Wilkins, RDH, DMD, both Tufts alumnae and faculty emeritae, have made to the Tufts and dental medicine and education communities.

Tillman is clinical professor emerita of public health and community service and a leading champion of geriatric dentistry. She graduated from Simmons College in 1945, and from Tufts University School of Dental Medicine in 1949 and was a member of the Tufts faculty for more than 50 years. She has devoted her career to dental education and was instrumental in developing Tufts' geriatric dentistry program, which has grown to become a unique educational model, blending academic knowledge and community outreach to effectively serve a diverse aging population. Today, Tillman continues to serve Tufts in conjunction with the City of Boston's Elderly Commission to provide free dental screenings for the elderly and disabled in the greater Boston area.

Wilkins, who passed away this week at the age of 100, was clinical professor emerita of periodontology and a world expert on dental hygiene. She graduated from Simmons College in 1938, received a certificate from the Forsyth School of Dental Hygiene in 1939, graduated from Tufts University School of Dental Medicine in 1949, and went on to establish the Dental Hygiene Program at the University of Washington School of Dentistry. In 1959, she published what has become the definitive text on dental hygiene, Clinical Practice of the Dental Hygienist, now in its 12th edition. She returned to Tufts in 1966, where she stayed until her retirement in 2011. Wilkins received the American Dental Education Association's William J. Gies Award for Achievement by a Dental Educator in 2012 and the International College of Dentists Distinguished Service Award in 2013.

"Drs. Tillman and Wilkins enrolled in dental school when not even 2 percent of practicing dentists were women. Now, more than 50% of the dental students at Tufts are women," said Huw F. Thomas, BDS, MS, PhD, dean of Tufts University School of Dental Medicine. "Drs. Tillman and Wilkins have been leaders in dental medicine, at Tufts, in their communities, nationally and internationally."

"Dr. Tillman's ongoing work with the school and our local communities exemplifies the leadership principles of passion and humanitarianism that we embody in our students," he continued. "And Dr. Wilkins was a true pioneer, a devoted educator and a friend to everyone at Tufts University School of Dental Medicine."

Established in 1996, the Dean's Medal recognizes individuals who have demonstrated loyalty, service and generosity. It is the highest honor bestowed by the dean of a school at Tufts University and is awarded to individuals who have made significant contributions to their school and the greater community.

Whitening Promotion from Vista

Posted on Friday, December 16, 2016

The average dental practice performs 100 whitening procedures per year. At an industry average of $40 per kit, that equates to $4000 in whitening expenses annually.

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Announcing the “Multidisciplinary Treatment Solutions for Peri-Implantitis” Symposium 

Posted on Thursday, December 15, 2016

Geistlich Pharma North America, Inc. is proud to announce with their symposium sponsors an interactive and didactic program covering topics around peri-implantitis. This three-day event will feature a surgical and non-surgical hands-on workshop as well as general sessions from world-class presenters. Come learn from leaders in our industry with expertise on the prevention, diagnosis and treatment in peri-implantitis complications. This multidisciplinary symposium is for all dental professionals who are responsible or interested in the placement, maintenance, restoration and preservation of dental implants. 

We encourage you to learn more about this program by visiting the following website:

Global exchange of high-level ideas and information will be carried throughout the event. The hands-on workshops, on Friday, June 9th, will be limited in attendance and are sure to sell out quickly. These interactive programs will review “Non-Surgical Periodontal Therapy,” presented by Dr. Marisa Roncati of Italy and “Corrections for the Prevention and Management of Peri-Implant Diseases,” presented by Prof. Dr. Frank Schwarz of Germany. 

General Sessions will commence on Saturday, June 10th and Sunday, June 11th and will include topics such as: 

“Prevent Peri-Implantitis and Plan for Long- Term Success” presented by Dr. Myron Nevins - USA 

“Identifying Factors Associated with Peri-Implant Bone Loss” presented by Dr. Paul Rosen - USA 

“Surgical Regenerative Therapy of Peri-Implantitis” presented by Prof. Dr. Frank Schwarz - Germany 

“Managing Peri-Implant Disease From a Laser Perspective” presented by Dr. Sam Low - USA 

“Peri-Implant Diseases: Understanding Etiology and Risk” presented by Dr. Joseph Fiorellini - USA 

“A Regenerative Algorithm for the Treatment of Peri-Implantitis” presented by Dr. Stuart Froum - USA 

“Peri-Implantitis Associated with Machined or Rough Surfaces” presented by Dr. Massimo Simion - Italy 

“Peri-Implant Disease and the Restorative Dentist”, presented by Dr. Chandur Wadhwani - USA 

“Is it Possible to Restore the Complete Health Around Implants Affected from Peri-Implant Disease?” presented by Dr. Marco Ronda - Italy 

“What Role Does the Restoration of Implants Play in Peri-Implantitis?” presented by Dr. Stephen Chu - USA 

More details on the symposium can be found at We hope to see you there! 




NYU Dentistry Awarded 5-year $1.9 Million NIH Grant

Posted on Thursday, December 15, 2016

Dr. Rodrigo S. Lacruz, assistant professor of basic science and craniofacial biology at NYU College of Dentistry, has been awarded a 5-year, $1.9-million grant from the National Institute of Dental and Craniofacial Research (NIDCR) to study calcium control in dental enamel. The research seeks to gain a better understanding of the impact of calcium in enamel mineralization and of the physiological processes by which enamel crystals are formed, and ultimately to develop improved strategies for the prevention and treatment of dental caries.

"Changes in the concentration of calcium (Ca2+) within the cell and the physiological mechanisms by which these changes occur can trigger a number of processes. This change in Ca2+ concentration is modulated by ORAI1 and STIM1 proteins in enamel-forming epithelial cells known as ameloblasts," explains Dr. Lacruz. "Deficiencies in the normal functioning of these mechanisms result in amelogenesis imperfecta, a term that broadly describes types of abnormalities in enamel. These abnormalities can weaken the outer enamel surface and can lead to accumulation of oral bacteria in those weak spots, resulting in caries and other dental disease, including a massive breakdown of the enamel in patients with mutations in STIM1 and ORAI1 genes."

While the importance of Ca2+ influx via Ca2+ release-activated Ca2+ (CRAC) channels in ameloblasts is apparent in studies describing amelogenesis imperfecta, understanding how CRAC channels modulate enamel development and mineralization is limited, as animals lacking Stim1 and Orai1 die soon after birth. To address this challenge, Dr. Lacruz, in collaboration with Dr. Stefan Feske, associate professor of pathology at the NYU School of Medicine, and Dr. David I. Yule, professor of pharmacology and physiology at the University of Rochester Medical Center, has developed and is studying several mouse models in which the genes Stim1 and Orai1 have been removed from a number of tissues of interest such as ameloblasts, sweat glands, and salivary glands. This localized deletion, known as conditional deletion, enables researchers to analyze the specific function of CRAC channels in these tissues without harming the remainder of the body's organs.

"Animal models enable us to look at the cells at different times and in different ways to assess the changes that occur when cells are deprived of calcium. In our case, we are interested in understanding what occurs when the enamel crystals themselves are also deprived of calcium," says Dr. Lacruz. He also plans to utilize animal models to understand the enamel problems experienced by Down syndrome (DS) patients, as they often present with a host of enamel deficiencies such as abnormal mineralization and thinner enamel. Of particular interest for his upcoming research is the protein-coding gene known as regulator of calcineurin (RCAN1), which is elevated in tissues including the brain, heart, and muscle of patients with DS, where its function in enamel is unknown.

The research undertaken by Dr. Lacruz and his team has the potential to significantly impact scientific understanding of how calcium contributes to enamel formation. Dental caries prevention relies heavily on knowing how enamel is formed so that clinical techniques can be developed to replicate these processes. According to Dr. Lacruz, "It is our hope that the data we obtain will motivate physicians to involve dental practitioners early on in the treatment of patient with DS or patients with mutations to CRAC channels genes because we have established links between these diseases and deficiencies in enamel."

Researchers Add to Evidence That Common Bacterial Cause of Gum Disease May Drive Rheumatoid Arthritis

Posted on Thursday, December 15, 2016

Investigators at Johns Hopkins report they have new evidence that a bacterium known to cause chronic inflammatory gum infections also triggers the inflammatory “autoimmune” response characteristic of chronic, joint-destroying rheumatoid arthritis (RA). The new findings have important implications for prevention and treatment of RA, say the researchers.

In a report on the work, published in the Dec. 14 edition of the journal Science Translational Medicine, the investigators say the common denominator they identified in periodontal disease and in many people with RA is Aggregatibacter actinomycetemcomitans. An infection with A actinomycetemcomitans appears to induce the production of citrullinated proteins, which are suspected of activating the immune system and driving the cascade of events leading to RA.

“This is like putting together the last few pieces of a complicated jigsaw puzzle that has been worked on for many years,” says Felipe Andrade, MD, PhD, the senior study investigator and associate professor of Medicine at the Johns Hopkins University School of Medicine.

“This research may be the closest we’ve come to uncovering the root cause of RA,” adds first author Maximilian F. Konig, M.D., a former Johns Hopkins University School of Medicine fellow now at Massachusetts General Hospital.

Medical investigators have observed a clinical association between periodontal disease and RA since the early 1900s, and over time, researchers have suspected that both diseases may be triggered by a common factor. In the last decade, studies have focused on a bacterium known as Porphyromonas gingivalis, found in patients with gum disease. However, while major efforts are currently ongoing to demonstrate that this bacterium causes RA by inducing citrullinated proteins, all attempts by this research team have failed to corroborate such a link, says Andrade. But his team has persisted on finding alternative bacterial drivers, he says, because of intriguing links between periodontal disease and RA.

For this study, the investigative team with expertise in periodontal microbiology, periodontal disease and RA began to search for a common denominator that may link both diseases. Initial clues came from the study’s analysis of periodontal samples, where they found that a similar process that had previously been observed in the joints of patients with RA was occurring in the gums of patients with periodontal disease. This common denominator is called hypercitrullination.

Andrade explains that citrullination happens naturally in everyone as a way to regulate the function of proteins. But in people with RA, this process becomes overactive, resulting in the abnormal accumulation of citrullinated proteins. This drives the production of antibodies against these proteins that create inflammation and attack a person’s own tissues, the hallmark of RA.

Among different bacteria associated with periodontal disease, the research team found that A actinomycetemcomitans was the only pathogen able to induce hypercitrullination in neutrophils, an immune white blood cell highly enriched with the peptidylarginine deiminase (PAD) enzymes required for citrullination. Neutrophils are the most abundant inflammatory cells found in the joints and the gums of patients with RA and periodontal disease, say the researchers. These cells have been studied for many years as the major source of hypercitrullination in RA.

A actinomycetemcomitans initiates hypercitrullination through the bacterial secretion of a toxin, leukotoxin A (LtxA), as a self-defense strategy to kill host immune cells. The toxin creates holes on the surface of neutrophils, allowing a flux of high amounts of calcium into the cell where concentrations are normally kept low. Since the PAD enzymes are activated with calcium, the abrupt exposure to high amounts of calcium overactivates these enzymes, generating hypercitrullination.

The researchers previously found that a similar type of pore-forming protein that was produced to kill pathogens by host immune cells was driving hypercitrullination in the joints of patients with RA. These findings point to a common mechanism that is poking holes on cells, which may be relevant to the initiation of RA and also when the disease is being established, says Andrade. 

As part of its study, the team developed a test using the bacterium and LtxA to detect antibodies against A actinomycetemcomitans in blood. Using 196 samples from a large study of patients with RA, the researchers found that almost half of the patients — 92 of 196 — had evidence of infection by A actinomycetemcomitans. These data were similar to patients, with periodontal disease with approximately 60% positivity, but quite different in healthy controls, who only had 11% of people positive for A actinomycetemcomitans. More strikingly, exposure to A actinomycetemcomitans was a major determinant in the production of antibodies to citrullinated proteins in patients with genetic susceptibility to RA.

Konig cautioned that more than 50% of the study participants who had RA had no evidence of infection with A actinomycetemcomitans, which, he says, may indicate that other bacteria in the gut, lung or elsewhere could be using a similar mechanism to induce hypercitrullination.

Andrade further cautions that his team’s study only looked at patients at a single point in time with established RA, and that to prove cause and effect of A actinomycetemcomitans and RA, more research will be needed to track the potential role of the bacteria in the onset and evolution of the disease, which can span decades. “If we know more about the evolution of both combined, perhaps we could prevent rather than just intervene.”

An estimated 1.5 million people nationwide live with rheumatoid arthritis, according to the Centers for Disease Control and Prevention. Current treatments with steroids, immunotherapy drugs and physical therapy help some by reducing or slowing the crippling and painful joint deformities, but not in all patients. The exploration of alternative treatment options is necessary.

Additional Researchers from Johns Hopkins included Kevon Sampson and Antony Rosen, MD.

This research was funded by the Jerome L. Greene Foundation, the Donald B. and Dorothy L. Stabler Foundation, Fundación Bechara, Rheumatology Research Foundation, National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS) under grant numbers R01AR069569 and AR050026-01, the National Institute of Dental and Craniofacial Research (NIDCR) under grant numbers DE021127-01 and R37 DE12354, and the Intramural Research Program of the NIDCR.


In Memoriam: Esther Wilkins, BS, RDH, DMD

Posted on Wednesday, December 14, 2016

Statement from the American Dental Hygienists’ Association

It is with sadness that we share the news that Esther Wilkins, BS, RDH, DMD, dental hygiene’s matriarch, died on Monday, Dec. 12. We had celebrated her hundredth birthday only three days before. Wilkins dedicated her life to advancing oral health care, and her commitment to dental hygiene will not be forgotten.

Wilkins was the author of the textbook, Clinical Practice of the Dental Hygienist, the first edition of which appeared in 1959. The 12th edition was published this year. More than 90 percent of the dental hygiene education programs in the world include it on the syllabus. Every edition has a differently colored cover, and the book is so iconic to dental hygienists that, amongst themselves, they identify the era in which they were educated by the color of the book they used.

Wilkins earned a certificate in dental hygiene from the Forsyth School for Dental Hygienists in Boston, Mass., in 1939. She worked in private practice and in a school clinic while pursuing a doctorate in dentistry, which she earned from Tufts School of Dental Medicine in 1949.

In 1950, Wilkins single-handedly established the University of Washington Dental Hygiene School Program, developing the curriculum and teaching most of the courses herself. She served as its director for more than 10 years, after which she returned to Tufts to obtain a specialty in periodontology in 1964. Following graduation, she served on the periodontology department faculty at Tufts, teaching periodontal instrumentation well into the 2000s.

Over the course of her lifetime, Wilkins developed and presented more than 750 continuing education courses for oral health care professionals and presented them in the United States, Canada and countries around the world. A consummate educator, she loved teaching and spending time with dental hygiene students. At professional meetings, including the ADHA Annual Conference, she always took time to speak and be photographed with students. A highlight of the Annual Conference was the student quiz program, Are You Smarter than Dr. Esther Wilkins?

In a 2005 interview with ADHA, Wilkins said, “If you are a student in an associate degree program, I encourage you to continue and pursue your bachelor’s degree. Then move on to your master’s degree. You must read, read, read, and keep up with the current research and literature.”

A longtime member of ADHA, Wilkins encouraged dental hygienists to join the organization and attend its meetings. “No man is an island,” she said, “and many dental hygienists work alone. They may not have another dental hygienist in the practice that they can talk with. So volunteer — there are many opportunities to volunteer in your local dental hygiene association and in community health.”

Wilkins is mourned by countless friends and colleagues around the world. 


Read Inside Dentistry's 2013 interview with Dr. Wilkins here

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